Precision medicine, also referred to as personalised medicine, is a clinical practise in which patients are given medications that are appropriate for them based on their genetic, environmental, and lifestyle factors. It is a method made possible by molecular diagnostics that differs from the traditional practise of treating every patient with the same illness with the same medication and dosage. In actuality, the activity isn't always quite fresh. Hippocrates, known as the "Father of Western Medicine," first saw it 2,500 years ago in ancient Greece. Hippocrates believed in the individuality of disease and the need to give "specific medications to special suffering," as a fascinating article by Sykiotis et al points out. He assessed a person's constitution, age, and physical characteristics, as well as the season, to determine how to, in a sense, prescribe Pharmacogenomics is a field of study that focuses on the relationship between genetic variations and how they affect drug response. Despite the frequent interchange of the terms "pharmacogenetics" and "pharmacogenomics," "pharmacogenetics" typically refers to the influence of a single gene on medication response. People have about 20,500 genes, and 99.5 percent of those genes are comparable, according to the Human Genome Project (HGP), which was completed in April 2003. The remaining 0.5 percent are variations that may be related to a person's eye colour, blood type, propensity toward specific diseases, etc. Single Nucleotide Polymorphism (SNP, pronounced "snip") is the most prevalent type of DNA sequence variation found in the human genome. Deletions, insertions, tandem repeats, inversions, and Copy Number Variations are examples of another type of version known as Structural Variations (SV) (CNV). The human genome has around eleven million SNPs, or one SNP every 1300 base pairs. SNPs serve as biologic markers that determine a person's response to specific medications and sensitivity to environmental risks. The medication also causes biochemical and physiological changes in the body. PD is the drug's mode of action and how it affects the body. It determines how well the medicine reacts with the target cells, such as heart tissue or neurons. In order that the drug to have the greatest potential impact and the fewest possible side effects on the patient, the drug developer calculates the problematic stability between PK and PD. Any understanding of Pharmacokinetics (PK), Pharmacodynamics (PD), and PGx is necessary to expand individualised dosage regimens for patients. Every medication that enters the body follows a certain process of absorption, distribution, metabolism, and excretion (ADME). The bulk of these approaches are added together to form PK, which determines how much of the drug is needed to reach the site of action for a successful therapeutic result. An individual's innate genetic polymorphism may cause a change in the stability of PK and PD, changing how the body and the drug (or its metabolites) interact with one another. PGx presents the promise that individuals may receive tailored prescription medications aided by knowledge of PGt tests, and the individual. the frequency of illness development. The likelihood that a condition may manifest or return The drug or substance's potential for benefit is at its highest. the recommended dosage. the type and volume of beneficial therapy responses. likelihood of medication toxicity.