Issues and Perspectives in Psoriasis Management


Issues and Perspectives in Psoriasis Management

Psoriasis is recognized as a chronic inflammatory autoimmune disease caused by genetics, the immune system and environmental factors with inherent aberrations in the skin in the form of either consistently excess proliferation of cellular constituents, particularly the epidermis, keratinocytes to hyperproliferate and perpetuate the disease process. Psoriasis varies in severity from small, localized patches to complete body coverage. These skin patches are typically red, dry, itchy and scaly. Humans lose five thousand million skin cells every day. How this colossal shedding process can occur without there being a break in the skin barrier is unraveled by the discovery of the unique shape (tetrakaidecahedron) and binding capability of epidermal cells providing a very tight, cohesive bond, explaining how skin maintains a barrier even when it is shedding [1]. It could also help us to understand what happens when it forms incorrectly, which could lead to conditions like psoriasis. Failure in the interlocking barrier between cells (the tight junctions), may partly explain why in psoriasis there is an overproduction of epidermal cells, causing thick patches of skin on the surface.

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